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Oncogenic viruses, such as the hepatitis C virus, infect animals and cause cancer. These viruses disrupt the normal regulation of the host cell cycle by adding oncogenes (genes that promote uncontrolled cell growth) or interfering with the production of genes that prevent cell development.

Oncogenic RNA infections incorporate, hepatitis C infection (HCV) and human T-cell lymphotropic infection 1 (HTLV-1).

Oncogenic DNA infections incorporate EBV, hepatitis B infection (HBV), human papillomavirus (HPV), human herpesvirus-8 (HHV-8), and Merkel cell polyomavirus (MCPyV).

Oncoviruses are delegated immediate or roundabout cancer-causing agents, albeit some cross-over exists between the qualifications. Direct cancer-causing infections have viral oncogenes that straightforwardly add to neoplastic cell change, though backhanded cancer-causing agents cause persistent irritation, which can prompt oncogenic change.

Oncogenic viruses are those that infect animals and cause cancer, such as the hepatitis C virus. They are capable of causing cancer. Cancer-causing viruses can be DNA or RNA viruses.

Cervical cancer produced by the human papillomavirus (HPV), liver cancer caused by the hepatitis B virus, T-cell leukaemia, and numerous kinds of lymphoma are all known to be linked to viral infections.

In this transmission electron micrograph, HPV, or human papillomavirus, features a bare icosahedral capsid and a double-stranded DNA genome that is embedded into the host DNA. The sexually transmitted virus is carcinogenic and can cause cervical cancer.

Oncogenic virus cores can be either RNA or DNA.